Wednesday, October 30, 2019

Application letter for a job Essay Example | Topics and Well Written Essays - 1000 words

Application letter for a job - Essay Example I am devoted to my studies and the steps that I have to take to obtain a future at Trinity Fields. I work very well in team situations and have always developed a good rapport with students and co-workers. During the past two years I have had several placements as a supply teacher in mainstream and special needs schools (both English and Welsh medium). During the Summer 2008 Term I was teaching at Ysgol Gymraeg Ynyswen. At the present moment, I am teaching a CPA Class at Ysgol Rhyd y Grug. I enjoy teaching different age groups and also various topics across the curriculum. I have had the experience of taking responsibility for a target group of pupils from Key Stage 1 and 2 that require extra help with their literacy and numeracy skills. I feel that this is a great opportunity for the pupils to develop their confidence in writing and also their oral skills. I intend to develop this by organising whole class, group and pair activities that will result in the development of the pupils linguistic skills. As a result of several and wide ranging linguistic activities and also satisfactory written work, children of all abilities will be given the chance to improve their language skills. It is my personal belief that no child should ever feel inadequate, inept, or forsaken. I strongly believe that every child has an important contribution to make in the classroom. They should be recognized, in a positive manner, for their contributions no matter how big or small these contributions may be. Students should feel accepted and comfortable at all times within the confines of my classroom. Differences will be praised, acknowledged and discussed within my class. I will recognize, include, and respect all students be they gifted, average, or categorically disabled, privileged, under-privileged, gay, lesbian, blue, or green. I will stress and enforce the importance of this acceptance

Sunday, October 27, 2019

African Trypanosomiasis Sleeping Sickness Biology Essay

African Trypanosomiasis Sleeping Sickness Biology Essay African trypanosomiasis also known as sleeping sickness is a wide spread parasitic disease (disease caused by organism that lives in or on another from which it obtains nourishment) that can be fatal if not treated. It is estimated by the World Health Organisation (WHO) that it has 450 000 cases each year, however in the past there have epidemics (a rapid spread or increase) such as between 1896 and 1906 where it is believed that 300 000 to 500 000 died from the disease. [1] African trypanosomiasis is common in the sub-Saharan region near rivers, lakes, in gallery forests and in Savannahs where the large brown tsetse flies are present. It occurs in these remote rural areas because the health systems are weak and because most of these areas depend on agriculture, fishing, animal husbandry or hunting so they are exposed to the tsetse flies. [2]The disease has been present in Africa for a minimum of 14 centuries with millions of people being affected by it. As you can see below (in figure 1) the distribution of trypanosomiasis in Africa comprises currently an area of 8 million km2 between 14 degrees North and 20 degrees South latitude. tryp_map.gif [Fig. 1] Distribution of human African trypanosomiasis. http://www.who.int/tdrold/dw/images/legend5.gifEpidemic http://www.who.int/tdrold/dw/images/legend6.gifHigh endemicity http://www.who.int/tdrold/dw/images/legend4.gifLow endemicity http://www.who.int/tdrold/dw/images/legend7.gifAt risk http://www.who.int/tdrold/dw/images/legend3.gifAbsence of the disease   As well as African trypanosomiasis also occurs in South America it is called the American trypanosomiasis or the Chagas disease however the organism causing that disease is different to the Tsetse flies. African trypanosomiasis is however more common than the South American version and it is estimated that around 50,000 to 70,000 people are currently infected with it and around 48,000 people died from it in 2008. [3] If, like most diseases, African Trypanosomiasis is diagnosed early there is a high chance of survival. There are no effective vaccines, and the drugs used to treat this disease are often toxic and usually have many side effects. Untreated cases have a 100 percent  mortality rate. [4] The extent of African Trypanosomiasis is shown more clearly when compared to other diseases and during epidemic periods prevalence reached 50% in several villages in the Democratic Republic of Congo, Angola and Southern Sudan. Sleeping sickness was the first or second greatest cause of mortality in those communities, ahead of even HIV/AIDS. [5] There are two types of African trypanosomiasis which are common in humans. The first of the two sub species is trypanosomiasis brucei gambiense which causes a slow chronic trypanosomiasis in humans. This mostly occurs in central and western Africa, where humans are thought to be the primary target. The second is T. brucei rhodesiense and this causes a rapid onset of trypanosomiasis in humans and this is most common in southern and eastern Africa, where animals are the primary target. Tsetse flies are large flies which can be easily misinterpreted for a housefly but can be distinguished by various characteristics. These flies cause human sleeping sickness and animal trypanosomiasis (or nagana) as well as other diseases and its estimated it kills around 250,000 to 300,000 people a year. Tsetse flies are multivoltine (they have more than 2 generations per year) and there are 23 species of this fly existent today. Tsetse flies include all the species in the genus Glossina, which are generally placed in their own family, the Glossinidae. [6] AfrTryp_LifeCycle.gif[Fig. 2] [1] When a tsetse fly bites it takes blood from a human or animal host. [2] If the tsetse fly is infected it injects metacyclic trypomastigotes into skin tissue and the parasites enter the lymphatic system (part of the immune system) and pass into the bloodstream. Inside the host, they transform into bloodstream trypomastigotes and are carried to other parts of the body. [3] After this bloodstream trypomastigotes reach other blood fluids such as lymph, spinal fluid and continue the replication by binary fission (Asexual reproduction where parent cells divide into two equal parts.). [4/5] The entire life cycle of African Trypanosomes is in extracellular (outside the cells) stages. A tsetse fly becomes infected with bloodstream trypomastigotes when taking a blood meal on an infected host . [6] In the tsetse flies midgut (the mid section of the digestive tract), the parasites generate a cycle of trypomastigotes, they then multiply by binary fission and leave the midgut. [7] Thereafter the parasites transform into epimastigotes (a developmental stage in trypanosomes) [8] The epimastigotes reach the flys salivary glands and continue multiplication by binary fission . This cycle in the fly takes approximately 3 weeks to occur. http://www.icp.ucl.ac.be/~opperd/parasites/images/dia3.jpg[Fig.3] This picture shows an African male in the late stage of African trypanosomiasis Describe the biological processes involved in producing solutions to the problem You can spot African trypanosomiasis early if the following symptoms occur, and treating the disease early increases the chances of survival. Symptoms of sleeping sickness begin with fever, headaches, and joint pains. If untreated, the disease slowly overcomes the immune defences of the infected person, and symptoms spread to anaemia, cardiovascular and kidney disorders. The disease then enters a neurological phase when the parasite passes through the blood-brain barrier (BBB). The symptoms of the second phase is what gives the disease its name, sleeping sickness, besides confusion and reduced coordination, the sleep cycle is disturbed with a period of lower level consciousness and periods of inability to sleep progressing to daytime drowsiness and night-time insomnia. Without treatment, the disease is fatal, with progressive mental deterioration leading to coma and death. Damage caused in the neurological phase can be irreversible. [7] Stage 1 illness: 2 to 3 weeks later, Stage 1 disease develops as a result of the protozoa being carried through the blood and lymph circulation of the host. This phase of the illness is represented by a high fever that falls and rises again, also irritating rashes, and headaches may occur. The Gambiense form, in particular, includes extreme swelling of lymph tissue, with enlargement of both the spleen and liver, and greatly swollen lymph nodes. During this stage, the heart may be affected by a severe inflammatory reaction, particularly when the infection is caused by the Rhodesiense variety of trypanosomiasis. These symptoms usually occur because of our immune system trying to defeat the invading organism; this is because of the increasing activity of our cells damaging organs and tissues. This may cause some blood vessels to leak and as a result of this the protozoa may spread further around the body. The immune system responds so violently because the trypanosomes survive so well by quickly changing specific markers, which are the outer coats of unique proteins. These proteins stimulate the hosts immune system to produce immune cells which will specifically target the marker, allowing quick destruction of those cells bearing the markers. Trypanosomes, however, are able to express new markers at such a high rate of change that the hosts immune system is constantly trying to catch up. Stage 2 illness This involves the nervous system, especially Gambiense sleeping sickness, which has a phase in which the symptoms involve the brain. These symptoms are that the patients speech becomes slurred, mental processes slow, and the patient sits and stares for long periods of time, or sleeps. Other symptoms resemble Parkinsons disease, including imbalance when walking, slow and shuffling gait, trembling of the limbs, involuntary movements, muscle tightness, and increasing mental confusion. Untreated, these symptoms could eventually lead to coma and then to death. [8] There are various treatments for the disease but it depends on the how bad the disease has got and what stage it is at. Suramin, eflornithine, pentamidine, and several drugs which contain arsenic (a chemical which in higher doses is highly poisonous to humans), are all effective anti-trypanosomal agents. When the disease is in its early stages it is easier to control and its treatments are the most effective, the least toxic, so the earlier the identification of the disease, the better the prospect of a cure. At the second stage the treatments depend on drugs that can cross the blood and the brain, to reach the parasite, however these drugs are more toxic and therefore carry a risk with them. There are four drugs that have been registered for the treatment of sleeping sickness which are; pentamidine, melarsoprol, eflornithine and suramin. If the disease is diagnosed early, the chances of cure are high. The type of treatment depends on the phase of the disease: initial or neurological. During which the second stage of the trypanosome parasites reside in the cerebrospinal fluid (CSF) so success in the latter phase depends on having a drug that can cross the blood-brain barrier (BBB) to reach the parasite. Four drugs have been used until now. Without treatment, sleeping sickness will lead to death. Unfortunately, however, those medications effective against the Trypanosoma brucei complex protozoa all have significant potential side effects for the patient. An external file that holds a picture, illustration, etc. Object name is AFHS0604-0223Fig1.jpg Object name is AFHS0604-0223Fig1.jpg [Fig 4] Sleeping sickness in South-Eastern Uganda from 1905- 2001 As you can see in figure 5 there were a high number of deaths of people from Trypanosomiasis from 1905-1915 but even thought the number decreases there have still been a lot of cases in this small region in the past 30 years and the number has been predicted to increase from The First Stage Treatment Suramin: Suramin is a colourless derivative. The development of suramin followed observations that a number substances. Suramin has six negative charges at physiological pH, thereby preventing its diffusion across cell membranes and it could possibly be used against late stage trypanosomiasis because it does not cross the BBB. Suramin is generally considered the drug of choice for the early stages of human African trypanosomiasis, especially T. b. rhodesiense infections. Because of the size and charge of suramin it makes it unsuitable to have a specific transporter. Suramin easily binds to many proteins. At attainable levels, more than 75% of suramin is bound to proteins, which include low-density lipoproteins (LDL). LDL and transferin are taken up through a receptor-mediated endocytosis and suramin enters the parasite specifically found to LDL. Accumulation of the drug in trypanosomes is relatively slow. In some studies, suramin has interfered with the metabolism of trypanosomes in different ways: It hampers the receptor mediated uptake of LDL, the carrier of cholesterol which is required for parasite growth Suramin, which enters via receptor mediated endocytosis in association with LDL, is most likely to accumulate inside the lysosome. Several of the enzymes encountered by suramin on its way from the extracellular fluid to the secondary lysosomes become inhibited by suramin. Many glycolytic enzymes located inside the glycosome on the African trypanosome carry a high positive charge, and therefore are all inhibited by micro molar levels of suramin. Second Stage Treatment Eflornithine: Eflornithine is an off-white, odourless, powder; the compound is freely soluble in water and sparingly soluble in ethanol. It is used mainly as a backup drug for melarsoprol but now in places such as Uganda it has become the first line treatment for sleeping sickness. Adverse drug reactions during eflornithine therapy are frequent. Their occurrence and intensity increase with the duration of treatment and the severity of the general condition of the patient. Generally, adverse reactions to eflornithine are reversible after the end of treatment. Trypanosomes are more susceptible to the drug than human cells, possibly due to the slow turnover of the ODC enzyme (which is key in the biosynthesis of polyamines) in T. b. gambiense. Eflornithine can effectively inhibit ODC activity and use the supply of polyamines in trypanosomes, which bring them into a motionless state that makes them vulnerable to the hosts immune attack. Therefore, a sufficiently active immune system is required to achieve a cure. Additionally, eflornithine induces the differentiation of thin forms to larger forms which do not divide anymore and therefore become accessible to the immune system. [9] It was found in a study that the effectiveness of eflornithine was 98.7% on its patients which is a high cure rate. Explain how the processes used are appropriate in terms of producing effective solutions to address the problem As you can see a total of 6 patients died, 5 of them on the 14 day course therefore the 7 day course seems to be much more effective. The 7 day course seems to be cause less adverse events in general then the 14 day course. [Fig. 5] However concluding figure 6 we can see that the probability of cure is higher in the 14 day regime then the 7 day regime. So even though there are some side effects of the 14 day regime still has a greater chance of curing the disease.[Fig. 6] F1_medium.gif [Fig. 7] A total of 103 patients with second-stage disease were enrolled. Cure rates were 94.1% for the eflornithine group and 96.2% for the nifurtimox-eflornithine group. Drug reactions were frequent in both arms, and severe reactions affected 25.5% of patients in the eflornithine group and 9.6% of those in the nifurtimox-eflornithine group, resulting in 2 and 1 treatment suspensions, respectively. There was 1 death in the eflornithine arm and no deaths in the nifurtimox-eflornithine arm. [10] The nifurtimox-eflornithine combination appears to be a promising first-line therapy for second-stage sleeping sickness. [10] F1_medium.gif [Fig 8] shows the probability of an event free survival among 1055 patients; they were treated with eflornithine for newly diagnosed second stage human African trypanosomiasis in Ibba, Southern Sudan. 1756-3305-1-3-3.jpg[Fig. 9] The combined efforts of chemotherapy, systematic case detection and vector control led to a dramatic reduction in the incidence of sleeping sickness at the beginning of the 1960s as shown in figure 2. The grey bars represent the number of cases whilst the line graph shows the population screened. Identify 2 implications of the solution encountered Suramin As suramin can only be injected it causes problems as African Trypanosomiasis can be spread easily from the needles not bring cleaned properly. If the patients who dont have the disease are injecting with the same unclean needle as people who are diagnosed this would result in the non-infected patients being infected. Further problems of suramin are that it has many side effects which include abdominal pain, diarrhoea, metallic taste and joint pain. Less common side effects are loss of vision and swelling around eyes. It can even rarely cause fainting and death. The advantages are that many of these side effects can be stopped by using a small sample dose, 100 mg IV, to test any toxic reactions of the patients. Eflornithine The route of application makes financial difficulties due to the workload and the additional material needed. A recently investigated abbreviated course of 7 days was found to cause a high relapse rate for the treatment of new cases. The superior outcome reported for the treatment of relapses still has to be confirmed, since there were a very small number of participants in the study. If there was an oral form for the treatment of sleeping sickness it would be more appropriate as it would be cheaper and less time consuming. In a study the average cost of eflornithine per patient being 552.3 USD and the cost per patients life saved averaging 559.8 USD. [10] Eflornithine also has side effects which include acne stinging skin; headache; dry skin; itching; erythema (redness); dyspepsia(upset stomach or indigestion); alopecia Fortunately there are advantages of this drug as efforts for the developments of an oral form of eflornithine are currently being made under the World Health Organisation/World Bank special programme for research and training in tropical diseases. This drug is generally better tolerated than the first line drug melarsoprol. It was found in a study that the effectiveness of eflornithine was 98.7% on its patients which is very high and therefore if the illness is treated within its late stages, stage 2, the chances of survival are still high. Evaluate the benefits and risks to humans, organisms and the environment Benefits: The side effects of Suramin can be stopped by using a small sample dose, 100 mg IV, to test any toxic reactions of the patients, so this means it could An oral form of eflornithine is currently being made under the World Health Organisation/World Bank special programme for research and training in tropical diseases. This would lead to cheaper costs of to treat a patient; therefore more people can be treated and cured. Effectiveness of eflornithine is 98.7% on its patients which is very high and therefore if the illness is treated within its late stages, stage 2, the chances of survival are still high, so a lot less people die when using the treatment. Disadvantges: The average cost of eflornithine per patient being 552.3 USD is far too high to cure most of the people especially because the treatment mainly depends on donated money as the government of the Third World countries are too poor to afford the treatment, so by using this lots of people may never be treated and therefore there may be a rise in the death rate. As the treatment of patients decreases, if people donate less, the number of deaths from the disease will increase. Further disadvantages of the treatments are that they both carry many side effects, even though some of these side effects arent major, the lack of treatments in these poor countries could result in a gradual decrease in health over time. Also some of the side effects and over doses could lead to death or serious illnesses so it is important to administer the drugs carefully and correctly as the end result could be fatal. Discuss alternative views or solutions for the problem encountered Pentamidine: Pentamidine can be used instead of suramin for a first stage treatment of trypanosomiasis. It is medication with a very slow rate of diffusion across biological membranes and it only treats the T.B.G. form of African sleeping sickness in the first stage. It could be injected or inhaled by the patient. Because of poor GI absorption, the drug is administered IV/IM and is strongly bound to tissues, including spleen, liver, and kidney. Clinical improvement usually noted within 24 h of injection. Reported to have a >90% cure rate. Pentamidine does not penetrate the blood-brain barrier effectively and, therefore, does not treat CNS infection. [9] Fifty-eight patients in the early-late stage (early central nervous system involvement) of Trypanosoma brucei gambiense trypanosomiasis were treated with pentamidine and divided into four groups according to cerebrospinal fluid (CSF) indicators: white blood cell (WBC) count, protein level (CSF protein), and the presence or absence of trypanosomes. Group 1 consisted of eight patients with normal CSF WBC counts and CSF protein levels and trypanosomes in the CSF. Group 2 consisted of nine patients with elevated CSF WBC counts, normal level of CSF protein, and trypanosomes in the CSF. Group 3 consisted of 31 patients with high CSF WBC counts, normal CSF protein levels, but no trypanosomes in the CSF. Group 4 consisted of 10 patients with normal CSF WBC counts and CSF protein levels and trypanosomes demonstrated by CSF culture. Post-treatment follow-up of all patients for at least one year revealed three relapses. There were two deaths from diseases unrelated to trypanosomiasis or to the treatment protocol. Of these patients, 52 were followed for more than two years, the time necessary to confirm a complete cure, indicating a cure rate of 94%. Pentamidine is therefore effective in treating the early-late stage of T. b. gambiense trypanosomiasis, and is comparable with melarsoprol or eflornithine in terms of its tolerance and availability. [11] Side effects: Shortness of breath; closing of the throat; hives; swelling of the lips, face, or tongue; rash; or faintin;, bleeding or bruising; blurred vision; chest pain or irregular heart beat; chills; difficulty breathing dizziness, fainting spells or excessive tiredness; drastic appetite changes; mouth ulcers severe stomach pain; severe headache; seizureshttp://bryanking.net/wp-content/uploads/2009/01/hives.jpg [Fig 10] This picture shows hives which is a common side effect of pentamidine Melarsoprol: Melarsoprol contains an arsenic element with a reactive arsenoxide group. The presence of the arsenoxide enables the ability of lipid solubility and this allows passage across the BBB. Apart from its transport function, the arsenoxide group mediates in the killing of the parasites in the bodily fluid; CSF (Cerebrospinal fluid). Modification of the melarsoprol parent ring to generate other analogous compounds can have a significant impact on its trypanocidal efficacy. The trivalent arsenicals; melarsoprol, melarsen oxide and phenylarsen are highly active with a minimum inhibitory concentration. Usually the transport of melarsoprol into the trypanosome parasite is accomplished by purine tranporters. Purine transport is highly developed in trypanosomes as they do not synthesize nucleic acids and must directly acquire them from their hosts. Resistance The failure of melarsoprol to cure 10% of the late stage sleeping sickness patients possibly relates to the fact that these individuals accumulate levels of the drug in the brain. However, one study has indicated that the levels of drug are similar in the CSF of relapsing and non-relapsing patients, so parasites at other extravascular sites may be key to the treatment failure. In some regions, treatment failures have reached high levels up to 30%. Parasites retrieved from the patients with these treatment failures were less responsive to melarsoprol than parasites isolated from other foci. This clearly points to some form of mutation towards resistance. Indeed arsenic refractory parasites do possess an unusual amino purine transporter which accumulates melarsoprol and the loss of this transporter in the parasite leads to drug resistance. T. brucei contains several of the purine nucleoside transporter activities. [9] Melarsoprol is a good alternative and is used as the first line drug against Human African Trypanosomiasis (HAT) in many countries. This is because the drug has longer intervals between when the dosages should be taken so it requires fewer resources and is therefore cheaper. However melarsoprol is a highly dangerous treatment, only administered by injection under the supervision of a physician. It causes a range of side effects, among them convulsions, fever, unconsciousness etc. It is fatal in and of itself in approx. 10% of the cases. Meanwhile eflornithine is a modern and far less dangerous treatment for HAT but it is expensive not widely available in the market and the money for supplies are usually dependable on donations. Melarsoprol is a good alternative and is used as the first line drug against Human African Trypanosomiasis (HAT) in many countries. This is because the drug has longer intervals between when the dosages should be taken so it requires fewer resources and is therefore cheaper. However melarsoprol is a highly dangerous treatment, only administered by injection under the supervision of a physician. It causes a range of side effects, among them convulsions, fever, unconsciousness etc. It is fatal in and of itself in approx. 10% of the cases. Meanwhile eflornithine is a modern and far less dangerous treatment for HAT but it is expensive not widely available in the market and the money for supplies are usually dependable on donations.

Friday, October 25, 2019

project proposal :: essays research papers

Proposal Essay   Ã‚  Ã‚  Ã‚  Ã‚  For this project, I chose to select the option of making an artist’s book. I have always found the portrayal of written words and pictures quite fascinating. Some have each page carefully worked out with decorative motifs varying from page to page. Some have fancy covers or style of binding that screams â€Å"pick-me-up!† But the type of books that fascinate me the most are those that are dedicated to the art of photography. I proposed for this book to be created as a photographic reference towards life. I wanted to create something that had meaning. I wanted it to tell a story, appeal to the eye as well as the mind. I chose to entitle the book ‘ABC’s of Life.’ Yes, I know that sounds kind of cheeseball, but a fancy name wouldn't get the point across.   Ã‚  Ã‚  Ã‚  Ã‚  This book was a challenge I hadn't ever taken part in and it was quite a bit of work. Each page in the book is designed as a black and white polaroid photograph. I wanted to capture the essence of spontaniality that is often found in life. I had to devise a list of words from a-z, each letter representing an aspect of life. For A, anger, an emotion used almost daily now-a-days. B, beauty, for it is seen in the eyes of all women and children. C, compassion, which is sometimes hard to come by, but no doubt in our lives. D, drugs, since we are all faced with them at one point or another. E, exotic, as whatever is one of a kind facinates us. F, fantasy, as we all wish we had something we don't. G, girlfriends, it's what makes worth living. H, hate, an emotion to be reckoned with. I, ironic, as some things are just too odd. J, journey, we are all headed somewhere, right? K, kiss, for when a yummy chocolate kiss makes your day. L, laughter, if we didn't have that we'd a ll be grumpy people. M, majestic, for all the sites you may see along the way. N, nocturnal, as some of us are people of the night. O, oxygen, what we take in every moment of every day. P, passion, it is what can make us whole. Q, questionable, as things aren't always what they appear to be. R, religion, can be what forms a persons life. S, sex, a bond that can unite and destroy.

Thursday, October 24, 2019

In Jane Harrisons play Stolen Essay

In Jane Harrison’s play, ‘Stolen’, the characters of Ruby, Anne and Jimmy are utilised in order to position the audience to feel sympathetic towards those affected by the ‘Stolen Generation’. Through her plot Harrison is able to demonstrate the pain faced by the characters. Furthermore, through her script, she is also able to show the mental disintegration of the characters throughout time. Therefore, it is imperative to examine the ways in which she has used these particular facets of her play in order to rouse the emotions of the audience. Jane Harrison utilizes the script of the play ‘Stolen’ to position the audience to feel sympathy for Anne. Anne is an aboriginal female who, adopted at a young age by a white Australian family. Anne was chosen by the white couple because ‘she was by far the best’ (THE CHOSEN pg 7). Anne is seen to have a ‘good upbringing’ (THE CHOSEN pg 7) compared to the other characters in ‘Stolen’. She receives a ‘sense of security’ (THE CHOSEN pg 7) and ‘a good education’ (THE CHOSEN pg 7), but Harrison reveals to the audience that Anne has to confront problems that none of the other characters have to face. Later on in the play, Anne is confused when she asks the question ‘Am I Black or White?’ (AM I BLACK OR WHITE? pg 28). Anne is torn between her origin and the people she has been brought up with. Harrison demonstrates this theme of ‘not belonging anywhere ‘through the script. Phrases such as ‘We’ve given you everything’ (AM I BLACK OR WHITE? pg 28) opposed to ‘But we’re your real family’ (AM I BLACK OR WHITE? pg 28). Harrison creates a binary opposition between the aboriginals, Anne’s blood and race, and the white Australians. Anne is rejected from both families, thus being rejected from everyone she knows, not belonging anywhere. Therefore, Harrison presents her view to the audience that even though Anne was better of materially compared to the other characters she had to experience a different type of pain that the other characters in ‘Stolen’ do not have to encounter. Harrison portrays the message that all children of the ‘Stolen Generation’ suffered, physically and/or mentally. Harrison uses the character Ruby to show the audience how mentally affected a child from the ‘Stolen Generation’ can be. Ruby was taken away from her family at a young age, just like many other children of the ‘Stolen Generation’. Harrison positions the audience to see that Ruby had to go through hard times as a child in the orphanage. In the scene ‘UNSPOKEN ABUSE 1’ (pg 8), Ruby has come back from a weekend away with a white family, the other children are curious and ask Ruby â€Å"What else did ya do?† (UNSPOKEN ABUSE 1, pg 8), and Ruby replies with â€Å"Promised not to tell† (UNSPOKEN ABUSE 1, pg 8). The audience does not know what happened to Ruby on that weekend but by the language Harrison has used, it seems that whatever actions that occurred on that weekend had affected Ruby had changed her. Harrison shows the audience in ‘RUBY COMFORTING HER BABY’ (pg. 9), that Ruby was an ordinary girl who played ‘with her doll’ (RUBY COMFORTING HER BABY, pg. 9). This same scene also shows the audience the horrible memories that Ruby have in her young mind, as Ruby is nurturing her doll, she seems like she is pretending to be her own mother and the doll being her. Ruby tells her doll ‘I love you Ruby’ (RUBY COMFORTING HER BABY pg.9). When Ruby grows up and leaves the orphanage, she goes and works for a white family. One day, her family come to visit, but it is revealed to the audience that Ruby is mentally disabled and is not well. Ruby’s family want to take her home but Ruby replies â€Å"Don’t live in no home any more. I work for the Hardwick’s† (RUBY’S FAMILY COME TO VISIT, pg. 31). It is clearly shown that Ruby cannot see that those people are her family. Harrison displays to the audience that in Ruby’s mind, Ruby believes that she has no family, reinforcing the fact that the children of the ‘Stolen Generation’ suffered immensely. Harrison shows the audience throughout th e play, the downfall of Ruby’s mental state Jimmy is a character in the play ‘Stolen’, who is an aboriginal male who has experienced pain throughout the play and this is shown through the play with the Harrison’s use of the plot and script. The character Jimmy spent his childhood years in an orphanage. The audience see that Jimmy had been brought up with no parents, just like many of the other characters in ‘Stolen’. Like Ruby, Jimmy goes away with a white family for a weekend and comes back changed, more timid than before. Jimmy grows up and leaves the orphanage he enters a bar and some indigenous people recognised him as ‘Wajurri’ (JIMMY’S STORY, pg. 27), and they said they knew his mother. Jimmy comes to visit her but before he can meet his mother, she dies. Jimmy is so devastated that he kills himself to finally ‘go meet my mother’ (SANDY AT THE END OF THE ROAD, pg. 36). The characters Ruby, Jimmy and Anne have many similarities and differences throughout the play and Harrison uses these similarities and differences through the script and plot. Both Ruby and Anne eventually meet their families. At the end of the play, Anne is accepted by her aboriginal and white families, feeling a sense of belonging towards both of her families. The audience see that Ruby is become completely insane and even though she faces her family, she does not believe that it is her family and goes back to work. Jimmy does not get to meet his family. When Jimmy is grown up, he discovers that his mother is alive, but when he comes to visit her, he finds out that she has died. This was extremely traumatic for Jimmy, and he couldn’t endure the grief and resorts to ending his own life. Jimmy, Ruby and Anne all faced mentally enduring events at some point in their lives. But only the only happing ending out of these three characters is the one of Anne’s. Ruby’s end is a more tragic one, as she is permanently scarred from the events that have occurred to her in her life. Jimmy also suffers a tragic end as the he commits suicide. Harrison depicts the harshness and undergone by both of these characters. Ruby and Jimmy’s upbringing were very dissimilar in comparison to Anne’s, Anne living in a family that cared and provided for her, was contrasted by Harrison, towards Jimmy and Ruby’s lifestyle. Both Jimmy and Ruby were brought up in an orphanage with other aboriginal children whose families were also taken away from them. The play ‘Stolen’, written by Jane Harrison shows the audience the hardship undergone by the characters, Ruby, Jimmy and Anne. Though they are different in many aspects, these characters share the pain of not knowing where to belong and this is shown by Harrison puts forward this idea through her use of the plot and the script. WORKS CITED Harrison, Jane. Stolen. (3rd rev. Edition) Strawberry Hills: Currency Press, 2007

Wednesday, October 23, 2019

Realism Naturalism Modernism

During this literary period, notable African American writers contributed o this diverse array Of literature. African American writers of this period â€Å"were often engaged in bitter disagreement over the form and functions of African American expression, over the obligations of black writers to their publications, and even over how such publics were to be identified. † (94) By tackling issues that dealt with identity, sexuality, social tensions, internal confessions, gender and gender roles, etc. He writers of this period sought to give a certain social literary reality to this broad style of writing. Modernism depicts a strong internal break with tradition; style or event that aims to break with classical and traditional forms and attach modern ides. James Baldwin, a notable contributor to the realism, naturalism, and modernism era, pulled from personal experiences to formulate writings. â€Å"Line king the most intimate areas of his own experience with the broadest quest ions of national and global density. (390) With early life events such as his struggles with poverty, religious indoctrination, the estrangement from his step father, and identifying his sexuality, Baldwin managed to connect modern topics by showing how they connect with the traditional ideals adopted by society. In his literature he attempts to show, through his characters, how individuals adapt to the changing world as well as the changing times. Baldwin novel Giovanni Room, displays the literary subject of modernism through themes such as sexuality and sexual identity.Baldwin uses an array of characters in this novel to express how sexuality and sexual identity is neither homosexual nor heterosexual. It is neither black nor white. Baldwin purposely chooses not to disclose the race of the characters in this novel, in order to find a universal story. Romance and love can be experienced regardless of what someone identifies themselves as. The traditional ideals that some characters, especially the protagonist David, struggles with throughout the novel, show how societal labels effect certain ideas as either being considered â€Å"right† or â€Å"wrong'.The main conflict of Giovanni Room concerns Davit's sexual identity. Defining his sexuality or accepting that he likes men as well as women plagues the entire story. David wants to love women, because that is what society and family believes is morally correct. David is sexually attracted to men. By accepting this fact, in his mind, would devalue his masculinity as well as shame the values of his family and society. There are many reasons that contribute to Davit's internal conflict with his sexuality. The largest reason can be found in Davit's relationship with his father.In the novel, David has many flashbacks of his life. His mother passes when David is about five years old. Davit's father raises him and his Aunt Ellen lives with them as well. David describes his father as a calm man, very slow to ang er. An adolescent memory that David remembers greatly about his father are the cocktail parties that his father throws at their house. He says that his father is very sociable then, pouring drinks and flirting with women. One eight, Davit's father and his Aunt Ellen argue because his father returns home drunk and being out with women once again.His Aunt Ellen states that David is growing up and his father should be more careful about the activities he involves himself in around David. David, until this point, thinks nothing of his father and his relationships with other Women. After this point, he begins to look at his father differently and the women he encounters. Davit's father responds to Ellen during the argument, that all he wants is for David to grow up and be a man. This is one of the first instances where we see that being tit a woman or many women equates as the qualifier of being a man.This expression of sexuality that Davit's father states lead to David to battle with hi s own sexuality. In the beginning chapter of the novel, David befriends a guy by the name of Joey. The have a sleep over at Joey's house and begin to horse play and wrestle as boys do. The two go to bed, but are awaken by Joey saying that he has been bitten by a bed bug. David teases him and they play fight which leads the two to kiss and make love. David awakes in the morning admiring Joey's body. Suddenly after realizing how much smaller Joeys body compares o his and realizing that Joey is a boy, his feelings then change.David feeling ashamed thinks of what his own father would think as well as Joey's parents if the two were caught in bed together. He leaves and from that day on when he see's Joey he is cruel to him, picking on him to please his new group of friends as well as proving to himself in a way that he is not homosexual. David does not seem to be at peace with his sexuality. Even when in a relationship with his fiance Bella, she eventually tells him that he seems withdra wn especially while the two are being intimate with each other.At one mint in the novel, David meets Giovanni and he tells Giovanni that he has never been with a man. David knowing that he has told a lie says to himself that, ‘People are too various to be treated so lightly. Am too various to be trusted (5) Not only is David in denial about his sexuality, he does not even trust himself with someone's heart. There are times in the novel that after David has sex with men (Giovanni and Joey) he begins to think about his father as well as the other party's family. Imagining what they will think of two men being intimate with each other.This is another continuation of how societal views and family morals override David being at peace with his own sexuality. While in Paris, David involves himself with a community of homosexual men in which he considers his friends. These men take him out to bars as well as sometimes helping him financially. Through this group of friends he eventuall y meets his lover Giovanni. While at a bar one night, Davit's friend Jacques notices how comfortable Giovanni and David are together. He pulls David to the side and Jacques tells David that he looks like something in a vision.He tells him that this is an important day for him. ‘†You re lucky that what is happening to you now is happening now and not when you are forty, or something like that, when there would be no hope for you and you would simply be destroyed† (66). David pretends he doesn't know what is going on and Jacques presses the point by asking if he will write a letter to Hell to tell her about Giovanni. David tells Jacques that he finds Jacques life despicable and Jacques responds that he could say the same of Davit's life. In that David is not really being honest with whom he truly is inside and that is despicable itself.David asks if there is not some better way or him than to enjoy himself with young boys in the dark. Jacques tells him not to pretend that he too has not been with men in the dark. Jacques tells David that Giovanni affection for him should make him happy. Instead, he is ashamed. David says that he doesn't understand Giovanni kind of friendship, Jacques tells David, â€Å"Love him. Love him and let him love you. Do you think anything else under heaven really matters? † (98). Throughout the text Baldwin displays instances in which Davit's desires and sexuality are shaped not by him, but by society.Davit's character battles with what his ether will think of him, what society will see him as, and not by what his mind and body desire. Modernism explores ideals such as homosexuality and rids them of the traditional views that society sets to normalize.